Ganoderma Health Theory - I

Ganoderma mushrooms provide a number of benefits to health which can be easily understood with basic concepts of ganotherapy. These three basic concepts of ganoderma health theory also called ganotherapy are enough to understand the working and application of ganoderma. Ganoderma isn't a drug or medication based on modern health theory but its a herb, a natural traditional herb which works on natural theory of health. So, in order to understand how and where, we can find ganoderma's effectiveness, we have to understand its bare basics which are based on traditional view and are a part of alternative health which may or may not be agreeable to followers of modern health theory. There are so many health theories each based on some different principle and we can not judge one theory based on principles of another health theory.

Ganoderma's health theory called ganotherapy has some basic assumptions on how and where it works. These basic assumptions do not help in understanding the basis of ganoderma's working but they only throw a light on who could expect health benefits from ganoderma. One simple theory that I have or like to follow is 'Fill it and Forget it'. I don't think that discussing how, what, where and why of any alternative health theory or modern health theory helps in solving a health problem. Discussing and learning about these aspects only help us in understanding how it works and has nothing to do with how effective it can prove for health status of our body system. I think it is better to try it for some time and see the results, results will make it clear whether there is an underlying health theory in ganoderma or any other ganoderma based products. It will also clarify whether it is real gold or something artificially polished like gold.

There are so many aspects of health and health theories that it becomes difficult to conclude anything. I've noticed that over time a single product is reported to be good, then bad, then good for one part of body or another based on new researches. These researches are not meant for common man but for health professionals who understand there basics and what this actually meant. A common man is only perplexed with all these complexities. We cannot research on a product like a health professionals due to lack of skills or due to lack of time to investigate every detail. We only need well researched or proven health plan which is simplified and easy to follow. Anything which is result oriented and effective solves our problem. In case of ganoderma, it isn't a modern artificially generated product or a health product developed by modern man in light of latest modern health theories. Ganoderma is a well estabilished product with credentials that no one can deny, ganoderma is not a dot com bubble which has surfaced due to contemporary reasons. It is another issue that today in the modern world of schemes and scams, anything which gains popularity is immediately imitated by many with both real and fake products. Sometimes it becomes difficult to distinguish between real and fake as at times fake seems to be more attractive and effective then real. I think nature must learn some tricks from modern man and work on overall look and appearance of its own creation.

This is first part of Ganoderma Health Theory and discussion on Ganotherapy continues in second part of this article also published with this one.

Yogesh Bailwal has been promoting Ganoderma and Ganoderma based products to those who are seeking natural health in today's atmosphere of polluted environment and polluted lifestyle. He accidentally discovered ganoderma and its health benefits when he was going through a difficult time. He believes in ganoderma's natural way of working on our body system and thinks that it is a perfect solution for those who are unable to take care of there health due to illness, age, disability, working condition, habits or any other factor which prevent a person from following other natural ways to health like exercising, yoga, meditation, sports etc. He manages a website on ganoderma and natural ways to care for health, with Infowebservices.org.
Above article is the first part of Basic Concept of Ganoderma Health Theory-II also published with this article on Ganotherapy.

High Blood Pressure, Tips on Natural Ways to Lower Your High Blood Pressure

How you can, on a natural way, lower your blood pressure. In this document we give some tips to help you achieve your pressure decrease. The purpose of this document is not to replace your medication nor doctor but rather a general awareness among people the importance of a correct balanced diet. This document is meant for the European point of view and therefor only applicable for the European market.

Medical name

The medical term for high blood pressure is hypertension. We speak of hypertension when the systolic blood pressure is = or

Risk factor

High or elevated blood pressure is a major risk factor for getting a heart attack or a stroke. Many adults suffer from hypertension, it is estimated over 30%. But not everyone is aware of this and the risks this entails. Hypertension is called the "silent killer", and it is not a disease in the usual way of the word, but it is certainly a risk factor which is best taken into account. Causes

Apart from any congenital abnormalities, the cause of hypertension is often a combination of factors, including too much stress, inadequate diet, insufficient exercise, smoking or excessive alcohol. However, it is possible to lower the blood pressure when applying some changes in lifestyle.

4 Tips

It is of great importance a high pressure is detected and managed in time so a heart attack or stroke can be prevented. Medications help prevent high blood pressure but often do not take the cause away. 4 tips for your high pressure by removing the cause:

Use less salt and less sugar; Both salt and sugar raise blood pressure and also have a strengthening effect on each other. Significantly reduce both the message.
Eat less red meat; It is still not entirely clear why red meat has a pressure elevating effect, but it is sure. A less red meat diet, eating more fresh fruits and vegetables has a clearly measurable reductions in blood pressure.
Provide more exercise; Exercise and sports are good for our body. Rest rusts, that's for sure. Take the stairs instead of the elevator, or parking the car further away, telephoning standing, go dancing, an errand on a bike, find a new sporting hobby, there are plenty of opportunities to increase physical activity as you are aware, and your actively to reduce pressure.
Use products that have a beneficial effect on pressure; extra virgin olive oil, dark chocolate, green tea

New, Ganoderma Lucidum

Recently men discovered that there is a herb that can help you fight your hypertension on a natural way. It is a mushroom that listens to the name Ganoderma Lucidum. And although that mushroom is known for thousands of years in China for its medicinal properties and its Chinese name (Ling Zhi) means season with spiritual power is it only briefly in the interest of medical science.

One cause of hypertension is blood vessel constriction by the accumulation of fats and cholesterol in the blood vessel wall but also thrombosis. The blood vessel is so narrow and the heart must pump harder as the pressure increases. Now it has been discovered that the Triterpenes in the mushroom constriction of blood vessels to go through the stored fat and cholesterol reduction. Adenosine monophosphate (AMP) Ganoderma has the ability to solve thrombosis. The blood flows much easier so for the heart this means less need to work hard and so is therefore a natural way to prevent hypertension.

Recently available as a supplement in Ganoderma Tea, Coffee and Hot Chocolate and so you work on a healthier vascular system while enjoying tea, coffee or hot chocolate milk.

More information on Ganoderma Lucidum / Reishi you will find at: http://www.ganoderma.be/en/a-cholesterol-diet-can-you-lower-your-cholesterol-with-a-diet/

Researchers Investigate 'Healthy' Obesity Gene

Main Category: Obesity / Weight Loss / Fitness
Also Included In: Genetics;  Diabetes
Article Date: 05 Jun 2012 - 0:00 PDT Current ratings for:
'Researchers Investigate 'Healthy' Obesity Gene'

Why is it that some obese people are healthier than others? This was one of the main questions Dr. Chaodong Wu of the College of Agriculture and Life Sciences - Texas A&M University System - and a group of researchers tried to answer in a recent study.

The study, which will appear in a July issue of the Journal of Biological Chemistry, used genetically modified mice to investigate the genetic aspects of why some obese people do not develop certain medical problems typically associated with obesity, especially Type 2 diabetes.

Wu noted that Xin Guo, a Ph.D. candidate in the college's department of nutrition and food sciences, contributed significantly to the study.

"Previous research had indicated that a regulatory enzyme which is encoded by the gene PFKFB3 protects against diet-induced fat tissue inflammation and systemic insulin resistance," said Wu, who also has a Texas AgriLife Research appointment. "Increasing evidence shows that fat deposition, or amount, is not directly associated with the inflammation or insulin resistance in the development of obesity-related metabolic diseases."

Wu said the inducible 6-phosphorofructo-2-kinase (iPFK2) enzyme links metabolic and inflammatory responses and may underlie what he refers to as "healthy" obesity.

"While many obese people develop Type 2 diabetes, heart conditions and other chronic health problems associated with being significantly overweight, other obese people do not," he said. "And while obesity in general is not healthy, some obese people do not develop the diseases more commonly associated with a less-than-healthy diet. Furthermore, a number of thinner people may have the sort of health problems more typically associated with obesity."

Wu said he and the other researchers theorized that these diseases are associated with the cellular inflammatory response brought on by obesity.

"We also thought this gene could conceivably be targeted for use in the treatment of diabetes, especially Type 2, commonly associated with obesity," he said. "We wanted to find out what might happen to a subject if that particular gene was activated."

Wu and his fellow researchers used laboratory mice to explore the effect of a targeted adipocyte overexpression of the gene/enzyme combination on diet-induced inflammatory responses and insulin sensitivity.

"We were trying to find out what it is in adipose, or fat, tissue that may trigger a negative response that leads to disease - and how to modulate that response," he said. "In our study, we learned overexpression of the iPFK2 enzyme increases fat deposition, suppresses inflammatory responses and improves insulin sensitivity in both adipose and live tissues."

As an extension of this research, Wu said, it may be possible to identify a pharmacological agent or bioactive agent which may have the desired effect on this gene toward reducing obesity-related cellular inflammatory response.

"We're hoping that, as one of its outcomes, this research will help lead to finding bioactive compounds or some type of supplement that might be taken to help activate this gene toward the promotion of health," he said. "It would also be a good idea to compare and contrast this research with studies done on what constitutes a healthy diet and the effect of such a diet at a cellular level. "

Wu said that would allow for screening bioactive compounds in a healthy diet to determine to what degree these might be applicable for the treatment of disease brought on by unhealthy obesity in an animal model.

"As a further extension, one might study different types of obese people and try to isolate additional specific genes that determine a healthy versus an unhealthy obesity and find a way to modulate the expression of those genes toward disease prevention and health promotion," he said. "Once you find the link between the gene and the obese status of the individual, then you could work with experts in chemical research to produce or replicate whatever pharmacological or bioactive compound is needed to treat unhealthy obesity."

Wu said it is important to determine positively to what degree obesity as a health problem is due to a person's genetic makeup as it relates to their ability to store fat, as well as what type of fat - saturated or non-saturated - the individual may store.

"Fat composition is more important than fat deposition, or content," he said. "We know fat cells secrete some of their own bioactive compounds that we may be able to isolate and identify for use in promoting health."

Wu said it will be necessary to discover the role of certain genes in the composition and deposition of fats beyond what has already been identified as being stored in the adipose tissue of mice.

"Then we may be able to produce a dietary supplement or other bioactive compound that would have a positive health effect," he said. "This could be used as a targeted treatment for obesity-related diseases such as Type 2 diabetes in a way that would have limited or minimal side effects."

Article adapted by Medical News Today from original press release. Click 'references' tab above for source.
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Alzheimer's Disease Process May Be Disrupted By Antioxidant

Main Category: Alzheimer's / Dementia
Also Included In: Cholesterol
Article Date: 12 Apr 2012 - 0:00 PDT Current ratings for:
'Alzheimer's Disease Process May Be Disrupted By Antioxidant'

Alzheimer's disease (AD) is now the sixth leading cause of death among Americans, affecting nearly 1 in 8 people over the age of 65. There is currently no treatment that alters the course of this disease. However, an increasing amount of evidence suggests that changes in the way the body handles iron and other metals like copper and zinc may start years before the onset of AD symptoms. A new study shows that reducing iron levels in blood plasma may protect the brain from changes related to AD.

In the current study a group of investigators from led by Dr. Othman Ghribi, PhD, Associate Professor, Department of Pharmacology, Physiology, and Therapeutics, University of North Dakota School of Medicine and Health Sciences, rabbits were fed a high-cholesterol diet which caused them to accumulate plaques of a small protein called beta-amyloid (Aß). These plaques are toxic to neurons and central to the development of Alzheimer's disease. The rabbits also developed changes in tau protein, which is part of the skeleton of neurons. When this protein becomes heavily phosphorylated, the ability of neurons to conduct electrical signals is disrupted. Following treatment with a drug called deferiprone (an iron chelator), the iron level in the rabbits' blood plasma was reduced and the levels of both beta-amyloid and phosphorylated tau in the brain were returned to normal levels.

Another degenerative process in AD involves the production of reactive oxygen species (ROS) that can damage neurons in the brain. Deferiprone is also thought to suppress this reactive oxygen damage caused by free iron in the bloodstream, however in this study there was no difference in reactive oxygen species in the treated group. It appears that iron in the AD brain is located in the wrong places - in particular it accumulates to very high levels in the cores of beta-amyloid plaques and is very reactive in this setting.

According to Dr. Ghribi, "Our data show that treatment with the iron chelator deferiprone opposes several pathological events induced by a cholesterol-enriched diet...Deferiprone reduced the generation of Aß and lowered levels of tau phosphorylation." While there was no effect on ROS levels, he comments that "It is possible that a higher dose of deferiprone, or combination therapy of deferiprone together with an antioxidant to prevent ROS generation would more-fully protect against the deleterious effects of cholesterol-enriched diet that are relevant to AD pathology."

Noted expert on metals metabolism research on AD Ashley Bush, MD, PhD, Mental Health Research Institute, Melbourne, Australia, adds that "this research highlights the role of metal ions as key modulators for the toxic interactions of risk factors for Alzheimer's disease, in this case cholesterol. Drugs targeting these metal interactions hold promise as disease-modifying agents."

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Genetic Variant Linked To Obesity And Insulin Resistance

Main Category: Diabetes
Also Included In: Obesity / Weight Loss / Fitness;  Genetics
Article Date: 28 Jun 2012 - 0:00 PDT Current ratings for:
Genetic Variant Linked To Obesity And Insulin Resistance

A large study in people at risk of diabetes has found a direct association between the presence of a small genetic alteration in a hormone receptor and increased body fat and insulin resistance. The results, to be presented Tuesday at The Endocrine Society's 94th Annual Meeting in Houston, suggest an adverse role for a previously described genetic variant, the BclI polymorphism.

"Our findings support the idea that even small variations in hormone receptor sensitivity can have metabolic implications, such as obesity or diabetes," said co-author Bastiaan Havekes, MD, PhD, of Maastricht University Medical Center, Maastricht, the Netherlands.

"Endocrinologists should not just focus on hormone levels themselves. Taking into account hormone receptor sensitivity could help in better understanding hormone-mediated effects on metabolism," he said.

The inherited BclI polymorphism occurs in the gene encoding for the glucocorticoid receptor, which controls the actions of glucocorticoids, steroid hormones that affect every system in the body. This small variant makes the receptor more sensitive to glucocorticoids, resulting in greater effects with similar hormone levels, Havekes said.

The effects of this change appear to be similar to, although much smaller than, the excessive glucocorticoid exposure that can occur from certain medications or diseases, Havekes said. Such excess exposure can result in weight gain, especially around the abdomen, as well as in disturbed blood sugar metabolism. This exposure most often occurs from long-term use of prednisone or other glucocorticoid medications, which are widely used to treat inflammatory diseases or to suppress the immune system. It also can result from endocrine diseases such as Cushing's syndrome. Cushing's causes overproduction in the body of the glucocorticoid cortisol, often called the "stress hormone."

Patients in this study, however, did not have known excess exposure to glucocorticoids, according to Havekes. He and his co-investigators studied 1,228 adults who participated in one of two Dutch studies focusing on diabetes in the general population. More than half of the study participants had either prediabetes (23 percent) or Type 2 diabetes (33 percent). All subjects underwent genetic testing for the BclI polymorphism.

The researchers found that 519 subjects did not carry the alternative form of the gene, or G-allele, for the BclI polymorphism on either chromosome. Another 540 subjects were heterozygous carriers, meaning the G-allele was present on one of the two chromosomes. The remaining 169 subjects were homozygous carriers and therefore carried the G-allele on both chromosomes.

Those who had the BclI polymorphism on each chromosome had a significantly higher body mass index and larger waist and hip circumferences than did noncarriers or heterozygous carriers, the authors reported. This was reflected by greater insulin resistance, meaning that insulin is less effective at lowering blood glucose (blood sugar).

"Determining an individual's genetic risk profile for metabolic disease is of paramount importance to prevent development of cardiovascular diseases," he said. "Future studies concerning cardiovascular risk profiling should perhaps consider the BclI polymorphism."

Article adapted by Medical News Today from original press release. Click 'references' tab above for source.
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Ganoderma - Could It Help In The Treatment Of Cancer?

By definition, Ganoderma is a "mushroom" from the green and vibrant Mycetes plant kingdom where short and tiny fungi that are unable to manufacture their own food like green plants--through photosynthesis--thrive. Within the plant kingdom, Ganoderma Lucidum belongs to a superior grade. Of all the many edible fungi in the kingdom, the Ganoderma mushroom stands out in its significant medicinal values.

Although Ganoderma may seem trivial to most people for it leads a parasitic life by breaking down and surviving on nutrients of other plants and animals, there is more to this mushroom than meets the eye. Known as the "superior herb," Ganoderma Lucidum is a rare variety of mushroom credited with the highest medicinal qualities in the Ancient China and is also a subject of many researches by various universities and organizations in the modern times. Also known as "Linghzi" in China--which is considered as an auspicious medicine because of its medical efficacy--and "Reishi" in Japan--which is called, the 'God's Herb', Ganoderma's worldwide popularity as a powerful medicinal herb continues to increase and is now mass-produced to help combat one of the dreaded diseases of today--cancer.

Ganoderma works best in the treatment of cancer because it helps cleanse the entire body from toxins while it strengthens immunity. It also contributes to the improvement of liver function while it stimulates the regeneration of liver cells--providing a very important supplement for those people who have liver cancer. Aside enhancing liver detoxification, the anti-cancer agents in Ganoderma which include Germanium and polysaccharides. The polysaccharide fraction of Ganoderma is responsible for its anti-tumor efficacy.

The indications for Ganoderma use in cancer include supplementation to reduce side-effects during radiotherapy or chemotherapy, to prolong survival, to improve quality of life, and to prevent occurrence or recurrence. While Ganoderma is used as a supplement during chemotherapy or radiotherapy to reduce side-effects such as loss of appetite, fatigue, hair loss, and risk of infection, it can also reduce the toxic and lessen the pains during the sessions particularly to cancer patients at terminal stages. Aside from helping the immune and endocrine system to prevent tumors, help improve the circulation and eliminate harmful free radicals, other qualities of Ganoderma that help in the treatment of cancer include the inhibition of DNA synthesis of the cancer cells, destroying the terminal enzyme activity of the tumor cells and restrain the spread of cancer cells.

Still, while Ganoderma has been recognized traditionally and scientifically as potentially useful in the treatment of cancer, there is still a notable discrepancy whether it can totally cure the Big "C" because of the lack of clinical trials demonstrating its efficacy. Despite these controversies, studies still suggest that though Ganoderma and its derivatives are not pharmaceuticals and have not undergone rigorous clinical trials to be tested against the disease, there are indirect clinical evidences to support its supplemental use in cancer.

Aside from being the biggest potential in curing cancer, the need to take Ganoderma is still indispensable for many illnesses are caused by the imbalance of body functions due to the accumulated toxins in the body.

Apart from removing the build-up of toxins, Ganoderma also protects and detoxifies the liver, prevents and cures heart diseases while it slows the aging process. It also has positive effect on insomnia, being a cure for high blood pressure and as effective treat diabetes. While it serves as a treatment for chronic bronchitis and asthma, Ganoderma also has effect on hyper susceptibility and is used as a beauty care medicine for the skin.

Terry Dunn is Webmaster of http://www.Ganoderma-Online.com - a Informational Resource about Ganoderma

Statins Cause Fatigue

Editor's Choice
Main Category: Statins
Also Included In: Cholesterol;  Sports Medicine / Fitness
Article Date: 14 Jun 2012 - 12:00 PDT Current ratings for:
Statins Cause Fatigue

Statin drugs are among the most widely used prescription drugs on the market, but now researchers say that the cholesterol-lowering drugs can cause fatigue and decreased energy upon exertion.

The study, published in Archives of Internal Medicine, was conducted by researchers at the University of California, San Diego, and involved more than 1,000 adults.

According to the researchers, doctors should take these findings in to consideration when prescribing these drugs.

The side effects of statins, particularly their effect on exercise, have gained increasing attention in recent months. Although some patients on statins report fatigue or decreased energy, the occurrence of fatigue-with-exertion or decreased energy in these patients has not been addressed in previous trials.

As a result, Beatrice Golomb, M.D., Ph.D., associate professor of medicine at UC San Diego School of Medicine and her team enrolled more than 1,000 adults from San Diego in order to investigate the occurrence of these side effects. Individuals with cardiovascular disease and diabetes were excluded.

Study participants were randomly assigned to receive either one of two statins at relatively low doses: pravastatin (Pravachol) at 40mg, or simvastatin (Zocor) at 20mg, or placebo.

The researchers chose these satins as they were the most fat-soluble and water-soluble of the statins, at doses expected to produce similar LDL ("bad cholesterol") reduction. The team state that the LDL reduction would be comparable to that with atorvastatin (Lipitor) at 10mg, or rosuvastatin (Crestor) at 2.5-5mg.

Study participants were asked to rate their energy and fatigue with exertion relation to baseline, on a 5-point scale, from "much worse" to "much better."

The researchers found that participants who received statins were considerably more likely to rate their energy and fatigue-with-exertion as "much worse", than those given placebo. According to the researchers. The effect seemed stronger in participants given simvastatin. Simvastatin reduced cholesterol levels significantly.

Golomb explained: "Side effects of statins generally rise with increasing dose, and these doses were modest by current standards. Yet occurrence of this problem was not rare - even at these doses, and particularly in women."

According to the researchers if, for example, 4 of 10 women on simvastatin reported worsened energy or exertional fatigue; 2 in 10 reported worsening in both, or rated either one as "much worse"; or if 1 in 10 participants rated energy and exertional fatigue as "much worse."

Golomb said:

"Energy is central to quality of life. It also predicts interest in activity. Exertional fatigue not only predicts actual participation in exercise, but both lower energy and greater exertional fatigue may signal triggering of mechanisms by which statins may adversely affect cell health."

The researchers note that findings from this study should be taken into consideration by doctors in risk-benefit determinations for statins, particularly for women, people aged 70-75(even if heart disease is present), and most patients without cardiovascular disease.

Written By Grace Rattue

Copyright: Medical News Today
Not to be reproduced without permission of Medical News Today Visit our statins section for the latest news on this subject. "Effects of Statins on Energy and Fatigue With Exertion: Results From a Randomized Controlled Trial"
Beatrice A. Golomb, MD, PhD; Marcella A. Evans, BS; Joel E. Dimsdale, MD; Halbert L. White, PhD
Archives of Internal Medicine, June 2012, doi:10.1001/archinternmed.2012.2171 Please use one of the following formats to cite this article in your essay, paper or report:

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